Things I’ve done since working as a physician
I enjoy seeing patients on the wards and in clinics, which I continue to do, but like Christmas pudding it’s possible to have too much of a good thing. I like variety, and a doctor whose only practice is seeing patients doesn’t have much influence on the next generations of doctors or how their service and hospital work. Easy to moan about ‘The Bloody Trust’.
Alongside doing an average of 25-30 hours a week of ‘being a doctor’ for the last 30 years, I’ve had a variety of roles in education and training (Sub-Dean for the Cambridge Clinical School; Chair of a National Postgraduate Specialist Training Committee) and hospital management (Clinical Lead for the Medical Admissions’ Unit; Clinical Lead for Renal Services; Deputy Medical Director).
I’ve recently stopped doing any formal roles in medical education or training and hospital management but have taken on being a Syndic of Cambridge University Press and a Member of Council of the Medical Defence Union.
Medical research
Describing the management of a patient having a heart attack, the handbook of medical emergencies I carried in my pocket as a houseman in 1981 states – and I’m not making this up - that ‘no specific therapy is required’. It would have been better if it had said ‘no specific therapy is available’. By a meandering path of incremental improvements, the standard of care in many cases now involves having a balloon inflated in the blocked coronary artery and a stent put in to keep it open, which – you probably won’t be surprised to learn – stops many people from dying. Nothing would have changed without medical research.
Success is not guaranteed. Patients can develop enormous bodily swelling in a kidney condition called the nephrotic syndrome. My first foray into medical research as a doctor in training involved trying to find out why nephrotic kidneys can’t get rid of fluid. I wrote a few scientific papers but didn’t discover anything of note.
A couple of years later I had another go. New experimental methods meant that molecular biology was allowing previously unapproachable problems to be tackled. For a few years I hitched my wagon to a very bright and driven fellow nephrologist who decided to try to work out how the kidneys know when to produce a hormone called epo (erythropoietin) that controls your blood count and stops you becoming anaemic, which happens to patients with kidney failure. It turned out that there isn’t anything special in the kidney: the mechanism is present in all cells, and it’s wonderful to now be able to prescribe drugs which interfere with it to treat anaemia in kidney patients. Peter (now Prof Sir Peter Ratcliffe) got a Nobel Prize in 2019. I learned a lot about how to approach biomedical problems from working with him.